Producing or stockpiling tabun was banned by the 1993 GA; Ethyl dimethylphosphoramidocyanidate; Dimethylaminoethoxy-cyanophosphine oxide; Dimethylamidoethoxyphosphoryl cyanide; Ethyl dimethylaminocyanophosphonate; Ethyl ester of dimethylphosphoroamidocyanidic acid; Ethyl phosphorodimethylamidocyanidate; Cyanodimethylaminoethoxyphosphine oxide; Dimethylaminoethodycyanophosphine oxide; EA1205InChI=1S/C5H11N2O2P/c1-4-9-10(8,5-6)7(2)3/h4H2,1-3H3InChI=1/C5H11N2O2P/c1-4-9-10(8,5-6)7(2)3/h4H2,1-3H3Except where otherwise noted, data are given for materials in their Lohs, KH: Synthetische Gifte. Tabun can become a vapor if heated.

Overview of tabun, including signs & symptoms, protection, & treatment ; Case Definition: Nerve Agents or Organophosphates Includes clinical description, lab criteria for diagnosis, & case classification. These, and the even more toxic soman (GD), were developed in 1944 and are members of the so-called G-agents.

The symptoms of exposure include: nervousness/restlessness, miosis (contraction of the pupil), rhinorrhea (runny nose), excessive salivation, dyspnea (difficulty in breathing due to bronchoconstriction/secretions), sweating, bradycardia (slow heartbeat), loss of consciousness, convulsions, flaccid paralysis, loss of bladder and bowel control, apnea (breathing stopped) and lung blisters.

People exposed to a low or moderate dose of tabun by inhalation, ingestion (swallowing), or skin absorption may experience some or all of the following symptoms within seconds to hours of exposure: MedicineNet does not provide medical advice, diagnosis or treatment. If the person who is poisoned cannot wake up, has a hard time breathing, or has convulsions, call 911 emergency services.For information on who to contact in an emergency, see the CDC website at The user should verify compliance of the cards with the relevant STATE or TERRITORY legislation before use. Poisoning by tabun resulted in a rapid deterioration of respiratory function and respiratory arrest within 5 min. It is classified as a nerve agent because it fatally interferes with normal functioning of the mammalian nervous system. Worek F(1), Kirchner T, Szinicz L. Author information: (1)Institut für Pharmakologie und Toxikologie, Akademie des Sanitäts- und Gesundheitswesens der Bundeswehr, Garching, Germany. CDC twenty four seven.

Capillary gas chromatographic analysis of nerve agents using large volume injections. Tabun was originally developed as a pesticide in Germany in 1936. Nerve agents are chemically similar to organophosphate pesticides and exert their … Recovery from tabun exposure is possible with treatment, but the antidotes available must be used quickly to be effective. The … Fluorescent detection of chemical warfare agents: Functional group specific ratiometric chemosensors. It is classified as a nerve agent because it fatally interferes with normal functioning of the mammalian nervous system. A fraction of an ounce (1 to 10 mL) of tabun on the skin can be fatal. Therefore, the best thing to do is avoid exposure: Leave the area where the tabun was released and get to fresh air. J Appl Toxicol 1994;14:317-31. However, nerve agents are much more potent than organophosphate pesticides. Producing or stockpiling tabun was banned by the 1993 GA; Ethyl dimethylphosphoramidocyanidate; Dimethylaminoethoxy-cyanophosphine oxide; Dimethylamidoethoxyphosphoryl cyanide; Ethyl dimethylaminocyanophosphonate; Ethyl ester of dimethylphosphoroamidocyanidic acid; Ethyl phosphorodimethylamidocyanidate; Cyanodimethylaminoethoxyphosphine oxide; Dimethylaminoethodycyanophosphine oxide; EA1205InChI=1S/C5H11N2O2P/c1-4-9-10(8,5-6)7(2)3/h4H2,1-3H3InChI=1/C5H11N2O2P/c1-4-9-10(8,5-6)7(2)3/h4H2,1-3H3Except where otherwise noted, data are given for materials in their Lohs, KH: Synthetische Gifte. Because tabun vapor is heavier than air, it will sink to low-lying areas and create a greater exposure hazard there. Aufl., 1967, Deutscher Militärverlag, Berlin (East).

The exact symptoms of overexposure are similar to those created by all nerve agents. Oxime doses of 25% or 5% of its LD(50) were used for pretreatment 15 min before tabun-poisoning and for treatment 1 min after tabun administration to mice. It is classified as a nerve agent because it fatally interferes with normal functioning of the mammalian nervous system.



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